WARNING TO THE US INTELLIGENCE СOMMUNITY. ILLICIT ISRAELI WAR AGAINST UNITED STATES THROUGH THE MOSSAD SHILLS IN CIA

May 14, 2020

by IRENE CAESAR, PH.D. News

SARS-2 (COVID-19) pandemic is a camouflaged — resurrected and recombinant — 1918 flu virus (H1N1) that caused the deadliest pandemic in the history of humankind (“Spanish Flu” killed an estimated 50 million people worldwide, including an estimated 675,000 people in the United States). The aggressive virulence factors (sustained human to human transmission), characteristic of 1918 virus, were specifically extracted from the 1918 virus and combined with various forms of human influenza and coronavirus. The 2009 H1N1 pandemic and COVID-19 pandemic (1918 virus camouflaged as “Coronavirus”) were meant to “celebrate” the 10th “anniversary” and the 100-year “anniversary” of the 1918 pandemic respectively.

It is established that the emergence of the 1957 H2N2 and 1968 H3N2 influenza A pandemic viruses was caused by the RECOMBINATION where new avian genome segments were imported into the backbone of 1918-descended H1N1 viruses (137), as well as the 2003 emergence of the pathogenic Fujian H3N2 influenza strain by interclade reassortment. Source:
Webby, R. J., and R. G. Webster. 2001. Emergence of influenza A viruses. Philos. Trans. R. Soc. Lond. B 3561817-1828
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2546865/

This means that the 1918 flu virus (H1N1) samples were carefully preserved and kept all these years, whatever CDC says about its recent digging out from the Alaska permafrost.

“Avian” or “swine” or “dogs” (coronavirus) specifics of the pandemics is explained by the biomaterial, in which the 1918 virus is grown. Coronavirus was specifically used to conceal that the SARS-1 and SARS-2 (COVID-19) pandemics were simply the heavily recombinant 1918 virus. I emphasize, specifically the 1918 virus had provided the sustained human to human transmission, i.e., high virulence of SARS-CoV-19 virus. This is called “GAIN OF FUNCTION” (GOF). The recombinant (man-made) nature of all the recent pandemics explains why CDC has “pre-pandemic vaccines”. Of course, if you make viruses (Pathogen) yourself, you have the vaccine (Antigen) automatically.

“The 2019 novel coronavirus, or “SARS-CoV-2″, was discovered because of Wuhan virus pneumonia cases in 2019, and was named by the World Health Organization on January 12, 2020. It belongs to the beta genera of the Coronaviridae family, together with SARS coronavirus in 2003 and MERS coronavirus in 2012. The alignment between SARS-CoV-2 and 2003 SARS CoV has about 70% sequence (some say 86%) similarity and 40% sequence similarity with MERS CoV. The coronavirus genome encodes a spike protein, an envelope protein, a membrane protein, and a nucleoprotein. Among them, spike protein is the most important surface membrane protein of coronavirus.” (source: https://sars-cov-2.creative-biolabs.com/sars-cov-2-2019-ncov-spike-protein-elisa-kit-329.htm). Remark: 70% sequence similarity and 40% sequence with SARS-1 and MERS is a proof of itself for the intentional “accelerated virus evolution”. “SARS-CoV-2 has all the same genetic equipment as the original SARS-CoV, which caused a global outbreak in 2003, but with around 6,000 mutations sprinkled around in the usual places where coronaviruses change. Think whole milk versus skim milk.” Source: https://www.snopes.com/news/2020/04/02/what-the-coronavirus-does-to-your-body-that-makes-it-so-deadly/

In the SARS-CoV-1 outbreak, the first Coronavirus virus, that was synthesized by CDC, was inefficiently transmitted by most infected people, so that, quarantine allowed the epidemic to be stopped before the virus could become fully established in humans. Now, its variation in the SARS-CoV-2, the second Coronavirus was artificially made to gain the ability to spread “efficiently”. The fairytale that this virus had “emerged naturally” is laughable, because the second version of SARS-CoV had suddenly emerged with GOF – GAIN OF FUNCTION. Evidently, somebody in CDC had worked hard to make the virus “spread more efficiently”. See: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2546865/#r137

This statement is confirmed by Dr. Fang Li of the Minnesota University in the important paper on the artificially GAINED OF FUNCTION SARS-CoV-1, analyzed below.

“Gain of Function” was objected by French scientist Dr. Simon Wain-Hobson of Pasteur Institute in Paris in 2014. He pointed that GOF is clearly the DURC = “Dual-Use Research of Concern”, i.e., the bioweapons:
https://www.ncbi.nlm.nih.gov/pubmed/25077136

In October 2014 the administration of US President Barack Obama banned GOF research on influenza, SARS, and MERS. Concerns over so-called “gain-of-function” (GOF) studies that make pathogens more potent or likely to spread in people erupted in 2011, when Kawaoka’s team and Ron Fouchier’s lab at Erasmus Medical Center in Rotterdam, the Netherlands, announced that they had modified the H5N1 bird flu virus to enable it to spread between ferrets (that is, to cross the barrier between species). The ban was lifted on December 19 2017, according to Science:
https://www.sciencemag.org/news/2017/12/nih-lifts-3-year-ban-funding-risky-virus-studies

Importantly, there should be “an intermediate host” for the Coronavirus to hop from snakes or bats to humans. Notably, the paper published by the US Federal government-funded researchers in the Nature Medicine on November 15, 2015 had proven that only the CHIMERIC SARS-like virus out of the surface spike protein of a coronavirus found in horseshoe bats, called SHC014, and the backbone of a SARS virus that could be grown in mice, CERTAINLY WITH OTHER ADDITIONS, can infect humans. See: “A SARS-like cluster of circulating bat coronaviruses shows potential for human emergence” by Vineet D Menachery, Boyd L Yount Jr, Kari Debbink, Sudhakar Agnihothram, Lisa E Gralinski, Jessica A Plante, Rachel L Graham, Trevor Scobey, Xing-Yi Ge, Eric F Donaldson, Scott H Randell, Antonio Lanzavecchia, Wayne A Marasco, Zhengli-Li Shi & Ralph S Baric. The research was jointly done by the University of North Carolina at Chapel Hill, USA; the Department of Cancer Immunology and AIDS, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts, USA; and the Chinese Academy of Sciences, Wuhan, China:
https://www.nature.com/articles/nm.3985.pdf?origin=ppub
https://www.nature.com/news/engineered-bat-virus-stirs-debate-over-risky-research-1.18787

Notably, “these certain other additions’ were made earlier, now forgotten in the public eye. In the paper “Structural Analysis of Major Species Barriers between Humans and Palm Civets for Severe Acute Respiratory Syndrome Coronavirus Infections” by Fang Li, published in J Virol. 2008 Jul, it is claimed that (1) “The major species barriers between humans and civets for SARS-CoV infections are the specific interactions between a defined receptor-binding domain (RBD) on a viral spike protein and its host receptor, angiotensin-converting enzyme 2 (ACE2); (2) to cross this inter-species barrier, “a chimeric ACE2 bearing the critical N-terminal helix from civet and the remaining peptidase domain from human was constructed, and it was shown that this construct has the same receptor activity as civet ACE2”. (3) Furthermore, “crystal structures of the chimeric ACE2 complexed with RBDs from various human and civet SARS-CoV strains were synthesized”. This means that the CROSS-SPECIES LINK was artificially synthesized to transmit SARS-CoV from a civet to a human. While a civet was a link from a bat to a human. Dr. Fang Li says that “the major species barrier for the transmission of SARS-CoV from a civet to a human WAS IN NATURE the INCOMPATIBILITY between four ACE2 residues (residues 31, 35, 38, and 353) in humans and two RBD residues (residues 479 and 487) in civets. That is why the NATURALLY-OCCURRING civet (animal) SARS-CoV virus was prevented from infecting humans due to “incompatible salt bridges at the hydrophobic virus/receptor interface”. Dr. Fang Li claims that after his manipulations with the SARS-CoV virus of civets, he was a success of trespassing this incompatibility “by eliminating unfavorable free charges at the interface through stepwise mutations at positions 479 and 487”. As the result, Dr. Fang Li says, the SARS-CoV virus of civets had gained “the sustained infectivity for human cells”. The newly SYNTHESIZED GOF SARS-CoV was submitted to the Protein Data Bank under accession numbers 3D0G (complex of chimeric ACE2 and hTor02 RBD), 3D0H (complex of chimeric ACE2 and cSz02 RBD), and 3D0I (complex of chimeric ACE2 and cGd05 RBD). Dr. Fang Li works for the Department of Pharmacology at the University of Minnesota Medical School, Minneapolis, Minnesota. Though the data on the Gained of Function SARS-CoV was filed by the University of Minnesota in July 2008 after the epidemic of SARS in 2002-2003, we can definitely claim that this 2002-2003 pandemic was itself a synthesized GOF binary biological warfare, though that time, the SARS-CoV-1 did not have enough human to human virulence. This was precisely corrected by the joint forces at the University of Minnesota. And, do, dr. Fang Li says that SARS-CoV-1 had “reemerged in Guangdong in 2003 to 2004, with four sporadic infections, no fatalities, and no subsequent human-to-human transmission. SARS has been absent in humans ever since”. This makes it clear that if not for the University of Minnesota efforts to keep SARS-CoV-1 highly virulent in the human-to-human transmission, we would not have gotten the present SARS-CoV-2 pandemic:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2446986/

Dr. Fang Li is a front man for Dr. Stephen C. Harrison who evidently wanted to stay in shadows for this controversial research. The research on the GAINED IN FUNCTION SARS-CoV-1 by Dr. Fang Li was funded by NIH (US National Institute of Health) grant CA-13202 to Stephen C. Harrison of Harvard. Dr. Stephen C. Harrison is the director of the Center for Molecular and Cellular Dynamics of Harvard Medical School, head of the Laboratory of Molecular Medicine at Boston Children’s Hospital, and investigator of the Howard Hughes Medical Institute. Remarkably, Stephen C. Harrison led the Structural Biology team at the Center for HIV/AIDS Vaccine Immunology (CHAVI) when it received National Institute of Allergy and Infectious Diseases (NIAID) funding of around $300 million to design and test the HIV vaccine. As is seen below, the HIV inserts play the major role in the GAINED OF FUNCTION SARS-CoV-2.

The research by Fang Li proves that there can be no direct transmission of SARS-CoV between bats and humans without an intermediate host. The argument that the closely related viruses in human can be a bridge for the SARS-CoV transmission from bats to humans does not hold, according to Dr. Fang Li research.

The Wuhan lab worked with the CLOSEST known relative of SARS-CoV-2, which is a bat coronavirus called RaTG13. The evolutionary virologist Edward Holmes, of the Charles Perkins Center and the Marie Bashir Institute for Infectious Diseases and Biosecurity at the University of Sydney, said in a statement from the Australian Media Center that “the level of genome sequence divergence between SARS-CoV-2 and RaTG13 is equivalent to an average of 50 years (and at least 20 years) of evolutionary change.” That means that in the wild, it would take about 50 years for these viruses to evolve to be as different as they are. Thus, we have a clear “accelerated virus evolution”. Nikolai Petrovsky of the College of Medicine and Public Health at Flinders claims that Bat coronaviruses can be cultured in lab dishes with cells that have the human ACE2 receptor, so that, over time, the virus will gain adaptations that let it efficiently bind to human receptors. Source: https://www.msn.com/en-us/health/medical/does-the-novel-coronavirus-have-any-links-to-a-high-security-lab-in-wuhan/ar-BB12QiM3

For recombination to occur, the two divergent viruses are made to infect the same organism simultaneously. So, SARS-CoV-2 has HIV inserts. The genetic mosaicism exists not only between divergent viruses, but also between viruses and bacteria. Thus, there certainly can be the recombination between the influenza virus H1N1 1918 and the SARS-CoV-2. That is why the Flu Vaccine Increases Coronavirus Risk 36% Says Military:

Flu Vaccine Increases Coronavirus Risk 36% Says Military Study

https://www.sciencedirect.com/science/article/pii/S0264410X19313647?via%3Dihub

And, in fact, CDC had published in its Volume 26, Number 6—June 2020 (retrieved on May 15th, 2020) the Research Letter “Co-infection with SARS-CoV-2 and Influenza A Virus in Patient with Pneumonia, China” by Xiaojing Wu, Ying Cai, Xu Huang, Xin Yu, Li Zhao, Fan Wang, Quanguo Li, Sichao Gu, Teng Xu, Yongjun Li, Binghuai Lu, and Qingyuan Zhan of China-Japan Friendship Hospital, Beijing; The Sixth Medical Center of PLA General Hospital, Beijing; Weifang No. 2 People’s Hospital, Weifang; Vision Medicals Co., Ltd., Guangzhou, all in China: https://wwwnc.cdc.gov/eid/article/26/6/20-0299_article

The coinfection, and, thus, mosaicism between SARS-CoV-2 and H1N1 (Spanish Flu 1918) is also confirmed by Dr. Judy Mikovits for the SARS-CoV-2 pandemic in the North Italy, please, see below.

In fact, “H” in H1N1 stands for haemagglutinin. The “H” in the Betacoronaviruses (like SARS-CoV-19) also stands for haemagglutinin. But in the case of H1N1, the hAEmagglutinin is written down as “HA” gene, while in the case of Betacoronviruses, the hAEmagglutinin is written down as “HE”. I claim that this confusion is intentional to hide the fact that SARS-CoV-19 is a cover up for the global biowar via the resurrected and GOF H1N1 Spanish Flu 2018 virus.

And, indeed, “researchers have drawn parallels between SARS-CoV-2 and the avian influenza viruses, noting that a protein called haemagglutinin in influenza is the equivalent of the SARS-CoV-2 spike protein and that furin activation sites may make these viruses so highly pathogenic”, — says Ana Sandoiu in “Medical News Today” on March 17, 2020: https://www.medicalnewstoday.com/articles/why-does-sars-cov-2-spread-so-easily

Ana Sandoiu emphasizes that SARS-CoV-2 is spreading much faster than SARS-CoV-1 of the 2002-2003 pandemic. In 2003, 8,098 SARS cases, with 774 deaths, occurred within 8 months. By contrast, within 2 months of the start of the SARS-CoV-2 outbreak, the new coronavirus infected more than 82,000 people, causing more than 2,800 deaths. And Ana Sandoiu claims that precisely the fact that SARS-CoV-2 shares the SAME haemagglutinin, as in H1N1, makes SARS-CoV-2 much more contagious than SARS-CoV-1. Thus, we have the crucial evidence of mosaicism between SARS-CoV-2 and H1N1 that goes beyond the mosaicism between SARS-CoV-2 and HIV, which both belong to the retroviruses. The stable mosaicism between SARS-CoV-2 and H1N1 is a conclusive evidence of engineering the SARS-CoV-2 as “GAIN OF FUNCTION” virus.

Ana Sandoiu continues: “Spike proteins are what coronaviruses use to bind to the membrane of the human cells that they infect. The binding process is activated by certain cell enzymes. SARS-CoV-2, however, has a specific structure that allows it to bind “at least 10 times more tightly than the corresponding spike protein of [SARS-CoV-1] to their common host cell receptor. This is due to the fact that the spike protein contains a site that recognizes and becomes activated by an enzyme called furin.” The “furin-like cleavage site” recently discovered in SARS-CoV-2 spike proteins may explain the viral life cycle and pathogenicity of the virus”. Moreover,“furin is a host-cell enzyme in various human organs, such as the liver, the lungs, and the small intestines. The fact that this enzyme resides in all of these human tissues means that the virus can potentially attack several organs at once.”

The furin recognition ability in SARS-CoV-2, absent in SARS-CoV-1, is also emphasized by Prof. Gary Whittaker at Cornell University, in Ithaca, New York, in the paper “Structural modeling of 2019-novel coronavirus (nCoV) spike protein reveals a proteolytically-sensitive activation loop as a distinguishing feature compared to SARS-CoV and related SARS-like coronaviruses” by Javier A. Jaimes, Nicole M. André, Jean K. Millet, Gary R. Whittaker. Prof. Gary Whittaker et al. says that the furin activation site sets the SARS-CoV-2 up very differently to SARS-CoV-1, in terms of its entry into cells, and the stability and virulence of SARS-CoV-2. Authors also confirm that, “since furin is highly expressed in lungs, an enveloped virus that infects the respiratory tract may successfully exploit this convertase to activate its surface glycoprotein”.

https://www.biorxiv.org/content/10.1101/2020.02.10.942185v1

This conclusion is confirmed by the paper “The spike glycoprotein of the new coronavirus 2019-nCoV contains a furin-like cleavage site absent in CoV of the same clade” by B.Coutarda C., VallebX.de Lamballeriea, B.Canardb, N.G.Seidahc, E.Decrolyb, published in Antiviral Research, Volume 176, April 2020. Paper also claims that the anti-2019-nCoV therapeutics should include the furin inhibitors. The authors specifically state that, that it is the specific form of hemagglutinin with the furin cleavage site that makes both the Influenza virus and the SARS-CoV-2 virus most virulent. Alike SARS-CoV-2, the highly pathogenic forms of influenza have a furin cleavage site, namely, the H5N1 hemagglutinin HA cleavage site, e.g., causing the hyper-virulence of the virus during the Hong Kong 1997 outbreak. Authors continue that the 2019-nCoV (SARS-CoV-2) S-protein sequence contains 12 additional nucleotides, which correspond to a canonical furin-like cleavage site. Authors specifically point out that this is the gain-of-function to the 2019-nCoV for efficient spreading in the human population compared to other lineage b betacoronaviruses.

https://www.sciencedirect.com/science/article/pii/S0166354220300528
Thus, the S protein in the SARS-CoV-2 was artificially generated, so that SARS-CoV-2 is a chimeric virus modified from the original SARS-CoV, with the addition of the desired S protein to make them more easily bind to human cells, thus amplifying their virulence and transmissibility. Also, SARS-CoV-2 exhibited an “uncanny similarity of unique inserts in the 2019-nCoV spike protein to HIV-1 gp120 and Gag.” The Indians discovered the 2019-nCoV (SARS-CoV-2) has four new sequences inserted — all of which can be found in HIV genetic sequences. The supposed HIV genetic insertions on SARS-CoV-2 gene are:
Insert 1: TNGTKR
Insert 2: HKNNKS
Insert 3: RYSL—TPGDSSG
Insert 4: QTNSPRRA: https://www.researchgate.net/publication/338957445_Uncanny_similarity_of_unique_inserts_in_the_2019-nCoV_spike_protein_to_HIV-1_gp120_and_Gag

Notably, the adaptation of HIV-1 to humans from chimpanzees was associated with a change in the p17 Gag protein, which may be involved in the specific targeting of the protein within the host cell cytoplasm. If SARS-CoV-2 virus has the Gag HIV insert, it might mean that precisely the Gag HIV insert is responsible for the adaptation of the highly contagious coronavirus from bats to civets and to humans.

Also, it is clear that HIV itself is a synthetic GOF virus, since its transfer from chimpanzees to humans had definitely happened through an intermediate host, which is “yet to be identified”, as it is claimed by the paper “Cross-Species Virus Transmission and the Emergence of New Epidemic Diseases” by
Colin R. Parrish, Edward C. Holmes, David M. Morens, Eun-Chung Park, Donald S. Burke, Charles H. Calisher, Catherine A. Laughlin, Linda J. Saif, and Peter Daszak, in Microbiol Mol Biol Rev. 2008 Sep. I bet that this “intermediate host yet to be identified” is CDC itself:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2546865/#r137

Analogously, “despite several proposed candidates, from snakes to pangolins to dogs, researchers have failed to find a clear “intermediate host” — an animal that would have served as a springboard for SARS-CoV-2 to jump from bats to humans”. The reason of why researchers have failed to find an “intermediate host” between bats and humans for SARS-CoV-2 is precisely the fact that SARS-CoV-2 was artificially synthesized, including by the efforts of Dr. Stephen C. Harrison of Harvard University and his protégé Dr. Fang Li of the Minnesota University.

Source: https://www.msn.com/en-us/health/medical/does-the-novel-coronavirus-have-any-links-to-a-high-security-lab-in-wuhan/ar-BB12QiM3

Dr. Judy Mikovits told The Epoch Times in her analysis and comparison of the virus of the SARS-Cov-2 (the virus that causes the COVID-19): “(it) apparently has genes that come from other human and other species including some envelope—the one from HIV.” Source: https://gulfnews.com/world/coronavirus-did-not-jump-from-wuhans-seafood-market-heres-the-evidence-1.1586936434717

In 2004, virologists demonstrated how retroviruses (specifically, immunodeficiency virus), pseudotyped with the SARS coronavirus spike protein, efficiently infect cells expressing angiotensin-converting enzyme 2 (ACE2) in humans. This research had demonstrated that “when using S-protein-pseudotyped SIV (in animals, similar to human HIV), the enzymatic activity of ACE2 made no contribution to S-protein-mediated infection”, meaning that ACE2 human receptors are not the main pathway. This means that HIV inserts in SARS-CoV-2 are the main pathway of infection. This paper “Retroviruses pseudotyped with the severe acute respiratory syndrome coronavirus spike protein efficiently infect cells expressing angiotensin-converting enzyme 2” . in J Virol. 2004 Oct. by Moore MJ1, Dorfman T, Li W, Wong SK, Li Y, Kuhn JH, Coderre J, Vasilieva N, Han Z, Greenough TC, Farzan M, Choe H. in J Virol. 2004 Oct. had in fact demonstrated that SARS-CoV is a CHIMERA of Coronavirus and HIV virus in animals (SIV). This combination, as the authors demonstrated, is “the codon optimization of the SARS-CoV S-protein gene” that “substantially enhanced S-protein expression”, that is, the virulence of SARS-CoV virus. They say: “Infection mediated by an S-protein variant whose cytoplasmic domain had been truncated and altered to include a fragment of the cytoplasmic tail of the human immunodeficiency virus type 1 envelope glycoprotein was, in both cases, substantially more efficient than that mediated by wild-type S protein.” The authors also stated that this was the additional “codon enhancement” to the in-itself enhancement of SARS-CoV virus to make it more virulent.

https://www.ncbi.nlm.nih.gov/pubmed/15367630

Also, this research had shown that a soluble and catalytically inactive form of ACE2 potently blocked infection by S-protein-pseudotyped retrovirus and by SARS-CoV. These results permit studies of SARS-CoV entry inhibitors without the use of live virus and suggest a candidate therapy for SARS. Source: https://www.ncbi.nlm.nih.gov/pubmed/15367630

A soluble and catalytically inactive form of ACE2 is sold at the moment by Creative Biolabs in the US, for example: https://sars-cov-2.creative-biolabs.com/stable-cell-line-ace2-cho-for-sars-cov-2-study.htm But this company grows ACE2 in the Chinese hamster ovary (CHO) cells, thus, destroying the Wave Optics of human chromosomes.

Recombination between Coronavirus and HIV (lentivirus) occurs since both of these viruses belong to the family of Retroviruses (Retroviridae) — single-stranded RNA viruses that produce reverse transcriptase by means of which DNA is produced using their RNA as a template and incorporated into the genome of infected cells, that are often tumorigenic.

Gp41 and gp120, the transmembrane glycoproteins, are the HIV “keys” to infecting human cells. Consequently, the recombinant-gp120-based vaccines were offered to the HIV-infected humans in 1990 by Philip Berman and colleagues in Nature. Again, If SARS-CoV-19 infects the host cell via the HIV mechanism of infection, then the ACE2 research is irrelevant for the possible cure:
https://www.nature.com/articles/d42859-018-00010-y

I refer to the blackop SARS-CoV-2 State Terrorism Global biowarfare in my article “WARNING TO THE US INTELLIGENCE СOMMUNITY. ILLICIT ISRAELI WAR AGAINST UNITED STATES THROUGH THE MOSSAD SHILLS IN CIA”:

https://irenecaesar.wordpress.com/2018/01/21/warning-to-the-us-intelligence-сommunity-illicit-israeli-war-against-united-states-through-the-mossad-shills-in-cia/

and in my February 25, 2018 article “THE BINARY BIOLOGICAL WAR APPROACHING”, published in March and April 2018 issues of the “Socialist Factor” Magazine, printed in Lucknow and London in 50,000 copies, and distributed to 240 embassies.

https://irenecaesar.wordpress.com/tag/bioelectronic-war/

It looks like that Mossad played the same role in the SARS-CoV-2 global bioware strike, as it played in 9/11. The Gained inatrlanta Function SARS-CoV-2 was secretly flown from the major CDC Influenza / SARS Biolab in Atlanta by the SINGLE Israeli plane through the Atlanta airport during the intentional Airport blackout, on December 20th 2017. In January 2018, DHS conducted the Biological Weapons drill, marking the beginning of preparations for the global biowarfare strike.

Those idiots and criminals, who had resurrected H1N1 1918-19 Spanish Flu virus, took their number of 60 millions killed worldwide by SARS-CoV-2 precisely from 50 millions killed by H1N1 1918-19 Spanish Flu Virus, in their mathematical model presented at the October 19, 2019 Event in NYC.